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Description

MAST, MEFIB, and MRE-NASH showed higher AUCs (0.785, 0.794, and 0.796, respectively) than did those of FAST, NFS, APRI, and FIB-4 (0.743, 0.751, 0.675, and 0.780, respectively) for predicting Fibro-MASH, although there was no definite statistical significance

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Previous studies have demonstrated that HSPs and their co-chaperones are involved in modulating the interaction between estrogen receptors and estradiol, as well as in regulating estrogen receptormediated cell proliferation [74, 75]

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This suggested that GSTP-silencing caused perturbation of mitochondrial homeostasis and consequently induced oxidative stress

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It has several clinical implications, including its role in ischemia

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All products for research use only

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