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Description

MBio 13(3):e03440-03421

Its proposed mechanism is unusual: Selank appears to modulate the expression of GABA-A receptor genes without binding the benzodiazepine site , which is offered as an explanation for anxiolytic activity that comes without sedation, tolerance, dependence or withdrawal [3]

D2 has an important role in pituitary and hypothalamic feedback (96)

Hormesis is featured with biphasic dose response, meaning that low doses of the stressor are neuroprotective, while high doses can exert a neurotoxic action in the brain