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Therefore, despite its known protective roles within mitochondria and possible interaction with NRF2 signaling, the overexpression and activation of TST have received limited attention as a potential therapeutic strategy to address diseases associated with mitochondrial dysfunction

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Although ROS contribute to oxidative stress and inflammation, they are also essential for normal physiological functions

Glucocorticoid regulation of adipocyte differentiation: hormonal triggering of the developmental program and induction of a differentiation-dependent gene