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In mice with myocardial hypertrophy induced by angiotensin II (ANGII), the expression of SLC7A11 decreased, and the inhibitory effect of SLC7A11 increased the contents of PTGS2, MDA and ROS, which aggravated cardiomyocyte hypertrophy

Differentiating this condition from primary psychotic disorders requires establishing the temporal relationship between mood and psychotic symptoms

MitoQ supplementation augments acute exercise-induced increases in muscle PGC1 mRNA and improves training-induced increases in peak power independent of mitochondrial content and function in untrained middle-aged men

HEK293 cells expressing Chac1-Myc were treated with MG132, lysosomal acidification inhibitors (Bafilomycin A1 (Baf) or Concanamycin A (CMA)), or the calpain I inhibitor N -Acetyl-L-leucyl-L-leucyl-L-norleucinal (ALLN)

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