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Explores the use of Homo-PROTACs for the chemical knockdown of cereblon, offering a novel strategy to selectively degrade cereblon and potentially enhance therapeutic approaches for diseases associated with its dysfunction

This may be one of the mechanisms underlying the effect of memantine in PD, as the K ATP channels on hippocampal neurons are composed of the same subunits as those on islet cells (Kir6.2 and SUR1) [186]

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