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Apparent mineralocorticoid excess syndrome arises when 11-hydroxysteroid dehydrogenase type 2 is deficient, allowing cortisol to activate mineralocorticoid receptors inappropriately and drive severe hypertension

Further, we also determined the effect of Glu supplementation on monoaminergic neurotransmission in brain and found that Glu supplementation enhances concentration of NA, DA and 5-HT via affecting their metabolism in brain

Think of them as clearing the exit route for fat the liver has already decided to export, not as the signal telling the liver to export fat in the first place

This analysis aims to further elucidate the mechanisms underlying BIPN development (Figure 2)

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