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On the other hand, SWS deficit has been linked to brain abnormalities, and is often reported in cases of schizophrenia and neurodevelopmental disorder [3]

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This is accompanied by the upregulation of pathways that mediate antigen presentation [e.g., major histocompatibility complex (MHC) II], phagocytosis related pathways such as C3, C4 and FCGR4, CD86 (Lagraoui et al., 2012), pathways that promote pro-inflammatory cytokines (e.g., IL-10, IL-6, IL-2, IFN-, IL-1) and chemokines expression (e.g., CXCL4, CCL4, CCL2)