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Because migration is highly dependent on actin polymerization, Grx2 knockdown would result in more globular actin (G-actin) production and prevent appropriate cell migration [37, 40]

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Jain DP, Dinakar YH, Kumar H, Jain R, Jain V

GPR30, stimulated by -estradiol, can be classified as a non-classical steroid receptor (G protein-coupled estrogen receptor, GPER)

6a, b, the SCs excellently adhered to the scaffolds and extended along the main axis of the fibers which was due to the significant effects of aligned nano-topography [55]