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However, this potential is shadowed by significant and unavoidable realities: a lack of human clinical data, an unapproved regulatory status, and valid theoretical safety concerns

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Depending on the context, specific mitomiRs can repress or activate gene expression: miR-2392 suppresses mtDNA transcription through AGO2, reducing OXPHOS and stimulating glycolysis [40, 41], whereas miR-1 and miR-1a-3p enhance mtCOX1 and mt-ND1 expression [37]

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