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Aterido A, Juli A, Ferrndiz C, Puig L, Fonseca E, Fernndez-Lpez E, et al

273,274,275,276,277,278 The detrimental role of CS in FM has been identified based on evidence from three aspects

Differential pathophysiological mechanisms between CHS and EHS While CHS and EHS share core pathogenic pathways (e.g., hyperthermia, inflammation, cell death), their mechanistic features differ significantly due to distinct patient demographics and triggers: Inflammatory kinetics EHS (predominant in young, active individuals) is characterized by rapid, exertion-amplified inflammatory responses

Overall survival and independent review of response in CheckMate 214 with 42-month follow-up: First-line nivolumab + ipilimumab (N+I) versus sunitinib (S) in patients (pts) with advanced renal cell carcinoma (aRCC)

Further experiments showed that TGR5 activation could induce the transformation of M1 (inflammatory) to M2 (anti-inflammatory) macrophages and inhibit the differentiation of Th1 and Th17 cells (72)