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Beneficial effects of Spirulina supplementation in the management of cardiovascular diseases

ALDH3A1 promotes cell proliferation via regulating energy metabolism reprogramming in NSCLC To investigate whether the reprogramming of energy metabolism was responsible for the progression of NSCLC, ALDH3A1 knockdown and overexpression cells were treated with 2-DG

A plaques result from the abnormal cleavage of amyloid precursor protein (APP) by betasecretase (BACE1) and gammasecretase, leading to the aggregation of A peptides, particularly A42, which is highly toxic to neurons (Hardy and Selkoe 2002)

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