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Overall, few low/intermediate-category patients moved to a higher fibrosis category at year 8

EA can activate ERK1/2 pathways and cyclin expression [119, 120] and neuroprotection can be obtained by proliferation of glial fibrillary acidic protein (GFAP)/vimentin/nestin-positive phenotypic astrocytes by enhancing BDNF expression in the peri-infarct cortex and striatum [121]

Unlocking the brains zinc code: implications for cognitive function and disease

Chapter 15, Keloid and Hypertrophic Scarring

The polarization of macrophages into M1 or M2 phenotypes is modulated by stimuli and signals from the inflammatory microenvironment [39]