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Since the known inhibitor only partially inhibits the HuR complex role in the cell, other works have also proposed genetic-based therapies, as well as in vivo delivery of silencing elements 87,88

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In accordance with previous results (17), we observe a unique and highly unusual fold for CTL1 among SLC proteins, with 10 transmembrane helices (M1M10), a large extracellular domain bridging M1 with M2, and three shorter amphipathic helices (AH1-3) parallel to the membrane that connect M4M5, M6M7, and M8M9, respectively (Fig 4A)