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IQGAP3 and IQGAP1 activate ERK-related cell proliferation and function as oncogenes, while IQGAP2 tends to inhibit tumor progression [14, 15]

MOTS-c is a mitochondrial-derived peptide that activates AMPK through direct mitochondrial signaling and nuclear translocation under metabolic stress, whereas metformin activates AMPK indirectly by inhibiting Complex I of the mitochondrial electron transport chain, and AICAR mimics AMP to trick the cell into AMPK activation