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Elevated ROS levels lead to endothelial dysfunction, which is a precursor to atherosclerosis

The historical perspective Initial clinical findings that the intestinal epithelium of IBD patients is more permeable to tracer molecules known to permeate through the paracellular space 28, 29, 30, 31 led to the hypothesis that an uncontrolled mucosal barrier may help promote IBD pathogenesis

BPC-157 route change: A patient who started with oral BPC-157 for GI support may transition to subcutaneous administration once GI tolerance is established, particularly if musculoskeletal recovery becomes a priority as exercise intensity increases

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