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Dysregulation of their expression or function modifies RBC behavior, hastening senescence and facilitating vascular disease (15, 21, 52)

A more comprehensive assessment of the bodys redox status can be achieved by measuring total antioxidant activity (TAA)

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When the damage surpasses the cells repair capacity, apoptotic pathways are activated, involving the expression of p53 and Bcl2 Associated X Protein (BAX, part of the Bcl-2 family proteins), the cytochrome c release, and the activation of caspase-3, -7 and -9, leading to apoptosis [2,19,24,30,31]