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Regulation of JNK signalling by GSTp

The discovery of SQORs unique cysteine trisulfide bond cofactor explains its remarkable catalytic efficiency ( 2 S levels, keeping them within an optimal physiological range: on the one hand, genetic defects (e.g., in Leigh syndrome) or CoQ deficiency that abolish SQOR activity can cause toxic H 2 S accumulation, inhibiting the mitochondrial respiratory chain and leading to severe energy failure

identified natural HLA-DR-associated peptides in autoimmune organs that will allow finding peptide-specific T cells in situ (18.2)

Kimoto A, Kasanuki K, Kumagai R, Shibata N, Ichimiya Y, Arai H

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