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discovered the tumor necrosis factor receptor type 1-associated DEATH domain (TRADD), which interacts with the intracellular structural domain of the TNF receptor 1 (TNFR1), in 1995, and suggested that TRADD is implicated in TNF-induced NF-B signaling

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Without these data, even a positive skin outcome would be difficult to attribute to a specific mechanism, and a safe cumulative-dose ceiling could not be defined