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Acknowledgments This work is part of the collaboration between the Department of Radiobiology and Molecular Genetics, VINA Institute of Nuclear Sciences - National Institute of the Republic of Serbia, University of Belgrade, Belgrade, Serbia, Clinic for Internal Medicine, Department of Endocrinology and Diabetes, Zemun Clinical Hospital, School of Medicine, University of Belgrade, Belgrade, Serbia, and KAUST

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*Correspondence: Dinesh Mahajan, [email protected] ORCID: Jishnu Sankar, orcid.org/0009-0007-7903-2484 Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers

Mutation of the kinase domain of LRRK2, G2019S, exacerbates ROS-induced dopaminergic neuronal death, and application of truncated LRRK2 reverses ROS accumulation and prevents morphological alteration of these neurons [52]

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