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Concurrently, hyperphosphorylation and its abnormal accumulation, coupled with impaired clearance, lead to the formation of NFTs, further compromising neuronal function

[2] The decline in GHK-Cu levels correlates temporally with increased risk of neurodegenerative conditions, and the peptide's antioxidant and anti-inflammatory gene expression profile is theoretically relevant to brain aging

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The fragment crystallizable gamma receptor (FcR) upregulates VEGF and IL-8 in RPE, whereas protease-activated receptor-1 (PAR-1) is a G-protein coupled receptor that aids in hemostasis and wound healing and is also involved in inflammatory processes