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RNA sequencing and pathway enrichment analysis show FOXO4-DRI significantly downregulates extracellular matrix-receptor interaction pathways, including fibronectin (Fn1), tenascin C (Tnc), and thrombospondin 2 (Thbs2), all critical components of fibrotic tissue architecture

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This editing predominantly occurs within inverted Alu elements, where ADAR1 binding competes with the RNA export factor STAU1, resulting in nuclear retention and diminished cytoplasmic translation of the edited transcripts

Although neutralizing antibodies have been proposed as a potential key mechanism of protection against many viral pathogens, antibodies mediate additional immune functions that may have both protective and pathological consequences

after let-7e overexpression, the cisplatin IC 50 values in A2780 and C13K cells decreased to 3.66 M and 9.66 M, respectively