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Abstract Objectives: Antioxidant defense status was investigated in HIV-infected patients by measuring serum selenium, erythrocyte glutathione peroxidase (GSH-Px) activity, plasma thiol (-SH) and glutathione (GSH) concentrations along with the assessment of the clinical stage and surrogate markers of HIV-disease

References were prepared according to the CHETAH R-package manual

Alternatively, GSH could have potentially prevented NRF2 activation through both KEAP1-independent mechanisms

As a powerful antioxidant, L-Glutathione protects cells from damage caused by oxidative stress and free radicals

The pool of fatty acids covalently bound to platelet proteins by thioester linkages can be altered by exogenously supplied fatty acids