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doi: 10.1038/srep42095 238 EspinozaJALenMACspedesPFGmezRSCanedo-MarroqunGRiquelmeSAet al

Notably, cerebral dysfunction has been identified as a potential extrahepatic symptom of NAFLD (284)

Although such difference in the inhibition behavior of EA toward FXIIIa-mediated dimerization and polymerization of fibrin(ogen) monomers is under investigation, the results indicate that the inhibition activity of EA toward the catalytic activity of human FXIIIa is physiologically relevant

This process is also critical in remodeling neurogenesis following ischemic strokes [51]

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