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Enhancement of thioredoxin/glutaredoxin-mediated L-cysteine synthesis from S -sulfocysteine increases L-cysteine production in Escherichia coli

While 7-HMR alone did not rescue dopaminergic cell loss in the acute toxin model (56), its ability to dampen neuroinflammation could be beneficial in a chronic setting or in combination with other therapies that promote neuron survival

Osmotic changes trigger the alterations of cell volume, which simultaneously activates cell volume recovery mechanisms and membrane channel proteins (Sadowska et al

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