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Together, these interconnected signaling cascades reshape transcriptional programs in fibroblasts, AECs, and endothelial cells, promoting myofibroblast differentiation, epithelial cell dysfunction, immune dysregulation, and excessive ECM deposition, which collectively drive the onset and progression of pulmonary fibrosis [33, 39] (Fig

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Alleviation of hypoxia stress induced oxidative damage, endoplasmic reticulum stress (ERS) and autophagy in grass carp ( Ctenopharyngodon idellu ) by TTO ( Melaleuca alternifolia essential oil)

Dimethyl fumarate (DMF), an FDA-approved drug prescribed for the treatment of multiple sclerosis ( Figure 7A ), allowing NRF2 to translocate to the nucleus and become transcriptionally active ( Figure 7 Indeed, our intracellular GSH detection assay revealed that both treatment with DMF and melatonin induced increases in ILC2 GSH levels ( Figure 7B )

To explore the lung regeneration process following successful fibrosis clearance, we mapped cellular profile by the scRNA-seq analysis of lungs between BLM+Saline and CD5/ LNP groups via the 10Genomics platform (Fig