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Riluzole remains the main approved therapy for ALS, despite yielding only modest survival gains through partial attenuation of glutamatergic excitotoxicity [7073]

This respiration rate is labeled OXPHOS capacity (alternatively known as NS-OXPHOS capacity)

In CON, the non-mitochondrial respiration rate ( Figure 9A ) remained at the baseline level throughout the experiment

Strikingly, blocking glutamine oxidation caused mitochondrial dysfunction only with ATMi, indicating a greater dependency upon glutamine (Figs

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