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Ge Lin, [email protected] These authors share first authorship This article was submitted to Developmental Physiology, a section of the journal Frontiers in Physiology Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers

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Mechanically, VPA effectively reduces the protein levels of renal injury marker KIM-1 and Bax, increases Bcl-2 expression, and inhibits the activity of caspase-3, therefore alleviating the kidney injury and apoptosis of RTECs induced by cisplatin