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[5] These enzyme families share no functional overlap with IgG catabolism

Mechanistically, multiple oncogenic signals, such as Myc and mTORC1, are involved in the activation of GLUD1, which is often accompanied by increased expression of other glutamine metabolism enzymes, such as GLS [142,143,144]

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Further research to explain the pathological consequences of these variants leading to this specific and consistent phenotype is required

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