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Moreover, the urinary excretion of isoprostane, a product of the nonenzymatic oxidation of arachidonic acid, and a marker of oxidative stress, was increased in vehicle-treated D 2 / mice and was almost completely normalized by etamicastat treatment (Fig

Transcriptomic analyses highlight the likely metabolic consequences of colonization of a cnidarian host by native or non-native Symbiodinium species

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S-adenosylmethionine (SAM), the methyl group donor substrate for DNMTs and HMTs, is formed in the one-carbon metabolism pathway, transformed to S-adenosylhomocysteine (SAH) during methylation, and then to homocysteine (HCys)