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In addition, confirming the efficacy of GSH treatment, a reduction in malondialdehyde, a marker of hepatic cell damage, was detected

CS exposure depleted ELF GSH levels by 4 hours from 100 M to 25 M, which was roughly the level where it remained until the end of the CS exposure (Figure 3A)

used angiotensin (Ang) II injection to simulate a hypertension-mediated cardiac remodeling model and found that ferroptosis occurred and that the protein expression of GPX4 and xCT was downregulated in the myocardium