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Longitudinal biodistribution data in chronic disease models remain sparse, and the persistent accumulation of non-biodegradable inorganic cores (e.g., Au, Ag, CeO) within microglia [156, 157, 179] or lysosomal compartments raises theoretical concerns regarding chronic low-grade inflammation or secondary lysosomal storage-like pathology over multi-year treatment horizons [159]

However, the PPV values observed in the external validation cohort remain higher than the general GDM prevalence, showing a relatively powerful risk stratification capability of the model

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[42], who documented significant increases in serum bilirubin concentration in previously sedentary postmenopausal women after being placed on an exercise regimen