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The preferential metastasis of ovarian cancer to lipid-rich tissues such as the omentum suggests that adipocyte-derived fatty acids are a significant source of energy for the rapid spreading and metastasis of ovarian cancer cells (36, 37)

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Preservation of mitochondrial function and cellular signaling integrity A key factor in the pathophysiology of polycystic ovarian syndrome (PCOS) is mitochondrial malfunction, which affects apoptotic control, steroidogenesis, and cellular energy metabolism [133]

Elevated MDA levels, a byproduct of lipid peroxidation, indicate an increase in lipid peroxidation in PCa patients, corroborating findings by Yossepowitch et al., which reported increased sensitivity to lipid peroxidation in the serum of PCa patients [12]

Immune dysregulation as a driver of idiopathic pulmonary fibrosis