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S13A), in line with other studies reporting upregulation necroptosis signaling components in human aneurysms[19, 79]

Multiple studies have demonstrated that MSC-Exos attenuate ferroptosis in chondrocytes by targeting key molecular axes

Beyond iron overload, whether iron depletion modulates ferroptosis thresholds by impairing mitochondrial respiratory chain complexesaffecting membrane potential and ROS generationrequires validation using conditional iron transporter knockout, iron chelators, and real-time mitochondrial monitoring (123, 124)

To determine whether this is the case, islets isolated from C57BL/6J mice ( n = 3 experiments) were either left untreated, exposed to alloxan (2 mM) for 30 min, or treated with alloxan following overnight incubation with THR-123 (10 M)

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