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Over time, mitochondrial damage accumulates, impairing their efficiency and releasing DAMPs into the cellular environment, which further stimulates inflammatory signalling, creating a selfperpetuating loop of mitochondrial dysfunction and OS, ultimately impairing energydependent wound repair processes such as cell migration and matrix deposition [52]

10.1016/j.neuroscience.2008.08.035 205 TalpadeD

It has been shown that astrocyte-mediated activation of transforming growth factor- -activated-kinase 1 (TAK1) in the mediobasal hypothalamus (MBH) mitigated anxiety-like behaviors (Zhao et al., 2021)

In PD, -syn aggregates also stimulate NLRP3 activation through mitochondrial stress and mtDNA release, exacerbating dopaminergic neuron degeneration [113, 114]

aureus have been categorized according to rep gene families (McCarthy and Lindsay, 2012) as well as the presence of functional genes ( S