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Abstract Parkinsons disease (PD) is one of the most common neurodegenerative disorders with a global burden of approximately 6.1 million patients

Consequently, in the context of targeting Acs as an approach to treating infections, the selective inhibition of microbial, parasitic, or fungal Acs enzymes relative to host ACSS2 would be likely to reduce potential adverse effects of those drugs

As a specialised protein scissor, it has the ability to break down such protein structures in a highly targeted manner [1]

reported modest therapeutic benefits of NAC treatment in both antisense-induced (NPC1ASO) and germline (Npc1/) knockout genetic mouse models of NPC1

Campisi (2005) proposed that specific gene upregulated during senescence and proteins translated from these genes may alter cellular functions and tissue structures