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From the diminished ROS buffering due to low cysteine/GSH [90], to slower immune cell proliferation from serine and glutamine paucity [10, 91], to weaker collagen scaffolding from glycine and proline shortfalls [93], and reduced perfusion and tissue anabolism from arginine/NO insufficiency [95] the elderly healing environment is at a distinct disadvantage

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These upstream signals converge on GjWRKY , a pivotal integrator coordinating ROS mitigation via GjPOD / GjGST activation, and osmotic adjustment through the ABA GjPP2C module and the proline-to-protein shift

It is a nonspecific marker but is also seen in other genetic disorders of glutathione metabolism [33,34]

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