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J Clin Oncol 2021 Gastrointestinal Cancers Symposium 39(suppl

Previous reports have suggested that cathepsin release from fractured lysosomes promotes caspase activation, leading to apoptotic cell death (1618)

To view a copy of this licence, visit About this article Cite this article Jarocki, M., Turek, K., Saczko, J

This is the theoretical basis for endogenously supplied gamma-glutamylcysteines ability to raise GSH levels, as being the product of the GCL enzyme, it can be taken up by cells intact, where it feeds directly into glutathione synthase to be converted to glutathione, effectively bypassing the dysfunctional GCL

The LC3-PE is then recruited to the autophagosome membrane