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Interestingly, accumulating evidence indicates that some NSAIDs, such as ibuprofen and diclofenac, exert additional effects beyond cyclooxygenase inhibition

Together, these findings necessitate further investigations to ascertain whether coordinated inhibition of NMDA receptors in GLP-1 receptor neurons modulates neuronal activity beyond canonical feeding regions, such as the mesolimbic dopaminergic reward pathway

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coined the term ferroptosis to describe this mode of cell death, which is characterized by its iron dependency and unique morphology, biochemistry, and genetics, distinguishing it from apoptosis and other forms of cell death [3]