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Mitochondria can release Ca 2+ into the cytosol via the mitochondrial permeability transition pore (mPTP) or the Na + /Ca 2+ exchanger [23, 24]

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Furthermore, they developed adenomyotic lesions that resembled the human disease (Huseby and Thurlow, 1982)

In addition, Fer-1, a ferroptosis inhibitor, upregulates the expression of GPX4 and downregulates the expression of ACSL4 (Wang et al., 2023)

Although EMDA gained extensive attention from investigators and might provide potential therapeutic benefit, current evidence does not support its use in the treatment of PD (Mehrsai et al., 2013)