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Therefore, inhibiting TLR signaling represents a novel therapeutic strategy for PD by modulating neuroinflammation (61)

1 2 "Inhibition of the human two-pore domain potassium channel, TREK-1, by fluoxetine and its metabolite norfluoxetine"

This suggests a shared stress or inflammatory response across these conditions, possibly mediated through the hypothalamic-pituitary-adrenal (HPA) axis or other stress-related pathways