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doi: 10.1016/j.atherosclerosis.2007.03.038 37

After injection into oleic acid-treated ARPE-19 cells, EVs, originating from organoid-derived human retinal progenitor cells, fused to its mitochondrial network and increased levels of mitochondrial fatty acid -oxidation-related proteins (CRAT and HADH) [100]

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and (2) most of the previous studies focused on the associations of GST M1 and T1 polymorphisms with only one type of acquired SNHL and neglected the fact that oxidative stress is the most important and common molecular mechanism of acquired SNHL