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Two endocannabinoids, anandamide and 2-arachidonoylglycerol, suppressed proliferation, induced apoptosis, deregulated the expression of cytoskeletal proteins (i.e., cytokeratins), upregulated lipid synthesis, and selectively activated the mitogen-activated protein kinase (MAPK) signaling pathway in human eccrine-sweat-gland-derived immortalized NCL-SG3 model cells

Third, multicenter clinical sample repositories should be established, and retrospective cohort studies should examine associations between ferroptosis-related biomarkers (e.g., GPX4, ACSL4) and patient outcomes