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While the involvement of various lipid species in the activity of Th17 cells remains a subject of debate, theres a growing consensus that imbalances in lipid metabolism can worsen the progression of autoimmune disorders

The elucidation of cuproptosis as a mechanism connecting inflammatory, metabolic, and mitochondrial damage to cardiomyocyte death opens new therapeutic avenues

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doi:10.1002/jlac.199419940618 Return to citation in text: [1] [2] Yoo, D