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This dysfunction can disrupt FA metabolism in astrocytes, potentially leading to neuronal damage from toxic FAs and exacerbating the progression of PD (Braidy et al., 2013)

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Researchers building inflammation protocol panels will find that GHK-Cus NF-kB mechanism is distinct from corticosteroid-based or COX inhibitor-based approaches, with a more selective and tissue-repair-compatible effect profile

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A.WatsonA.et al (2017)