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We explore the glucose divergence into the PPP and OXPHOS as well as the creation of ROS through mitochondrial leakage and the mitigation of ROS by the GSH redox system

PCS demonstrates extraordinary proficiency in activating renal inflammatory network architectures through complementary yet mechanistically distinct pathophysiological paradigms: it promotes ROS generation via NADPH oxidase activation cascades within renal tubular epithelial cellular matrices, simultaneously catalyzing JNK and p38 MAPK signaling pathway orchestration (Chang et al., 2020)

After protein removal by centrifugation (15,000g, 1 min), the supernatant was injected into the HPLC apparatus