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doi: 10.1002/hsr2.2019

Testosterone replacement has potential in reducing ADMA concentrations and increasing NO production in idiopathic hypotrophic hypogonadism [56]

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This therapeutic potential is primarily achieved by promoting angiogenesis, neurogenesis, and stem cell migration, which collectively contribute to a marked reduction in infarct volume and enhanced functional recovery in experimental models of cerebral ischemia [109, 110]