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Dr Luis Alameda was supported at the beginning of the preparation of the current work by the Swiss national Foundation (P2LAP3 171804)

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The anti-inflammatory effect of H

Oxidative stress directly damages lipids, proteins, and DNA in lung tissues, induces an inflammatory response, and also further accelerates the pathological process by inducing iron death in lung tissues [6]

It is characterized by iron-dependent lipid peroxide damage in mitochondria and a lack of glutathione peroxidase 4 (GPX4) and is distinct from apoptosis, autophagy, and necrosis (Dixon et al., 2012) (Figures 1, 2)