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Riboflavin seems to function as a proteasome inhibitor 43,45 , resulting in the decreased degradation of the NF-B inhibitor, phosphorylated-inhibitor kappa (P-IB), thus preventing nuclear translocation of NF-B and subsequent pro-inflammatory gene activation 46

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The issue might be particularly concerning for the pediatric population with chronic illnesses, as this would lead to accelerated deterioration of the disease with micronutritional deficiencies, or conversely, slowing down the deterioration process [135]

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