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The response to oxidative stress was found to be modulating 26 targets (CDC34, ATP5F1B, CREBBP, PPARGC1A, NTRK2, MAP2K1, PRKDC, PDE2A, MAP4K4, PRKAA1, NTRK3, ADORA1, PRKAA2, HDAC1, ALOX5, FOXO1, ESRRB, SIRT3, CDK1, RELA, EPHX2, NTRK1, HIF1A, SOD2, UBB, IGF1R) whereas the targets SIRT3, FOXO1, SOD2, and PPARGC1A which are also involved in the downregulation the ROS levels, and oxidative stress via mitochondrial process 48 , as shown in Fig

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E., Kalyani, D

Administered at a dose of 2.5 M, the drug had minimal effects on proliferating glioblastoma cells (LN229 and A172) while demonstrating significant senolytic properties against the same cell lines subjected to senescence induction by temozolomide [106]