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Targeted silencing or complete global knockout of KDM3A expression significantly mitigated oxidative stress, suppressed NF-B/P65 activation, reduced apoptosis and fibrosis, and improved cardiac function, which indicates the therapeutic potential of HDMi in diabetic cardiovascular injury [227]

Flow cytometry confirmed activation of P4-10bbz-expressing Jurkat cells upon exposure to recombinant/soluble GFR4 or co-culture with GFR4+ human MTC cell lines (TT, MZ-CRC1)